How so? There is no discussion of transmission.
Very preliminary theory and I am not an MD.
1. COVID-19 "utilizes
ACE-2 as a cellular entry receptor" by binding to it.
Quote taken from
https://www.rndsystems.com/resources/articles/ace-2-sars-receptor-identified2. "It is mainly expressed in vascular endothelial cells, the renal tubular epithelium, and in Leydig cells in the testes.
15,16 PCR analysis revealed that
ACE-2 is also expressed in the lung, kidney, and gastrointestinal tract, tissues shown to harbor SARS-CoV."
Quote taken from
https://www.rndsystems.com/resources/articles/ace-2-sars-receptor-identified3. "The results indicate that ACE2 highly expresses in renal tubular cells, Leydig cells and cells in seminiferous ducts in testis. Therefore, virus might directly bind to such ACE2 positive cells and damage the kidney and testicular tissue of patients."
Quote from
https://www.researchgate.net/publication/339243337_ACE2_Expression_in_Kidney_and_Testis_May_Cause_Kidney_and_Testis_Damage_After_2019-nCoV_Infection
4. "The seminiferous tubules are the site of the germination, maturation, and transportation of the sperm cells within the male testes."
quote from
https://www.innerbody.com/anatomy/male-reproductive/seminiferous-tubulesThis has, obviously, larger implications if true.